Cancer Metastasis - Related Genes (Hardcover, 2002 ed.)

Being diagnosed with cancer is devastating. But when the cancer cells have to spread to form secondary colonies, the prognosis for the patient is worse. If meaningful improvements in survival are to occur, then control of metastasis will be a foundation. Relatively little is known about the control of the metastatic process at the molecular level. This volume begins to explore our current knowledge regarding the underlying molecular and biochemical mechanisms controlling the metastatic phenotype. While all of the authors attempted to put their findings into a context for translation to the clinical situation, the state-of-the-art does not fully allow this. Nonetheless, we write these summaries of our work as an early effort toward that end. I am grateful to all of the authors who have contributed generously of their time and energies to make this volume a reality. To metastasize, neoplastic cells dissociate from the primary tumor, enter a circulatory compartment (typically lymphatics or blood vasculature), survive transport, arrest, exit the circulation and finally proliferate at a discontinuous site in response to local growth factors. Unless cells accomplish every step of the metastatic cascade, metastases cannot develop. The process is highly inefficient, i. e. ,

General

Imprint:	Springer-Verlag New York
Country of origin:	United States
Series:	Cancer Metastasis - Biology and Treatment, 3
Release date:	July 2002
First published:	2002
Editors:	D. R Welch
Dimensions:	235 x 155 x 17mm (L x W x T)
Format:	Hardcover
Pages:	270
Edition:	2002 ed.
ISBN-13:	978-1-4020-0522-0
Categories:	Books > Medicine > Clinical & internal medicine > Diseases & disorders > Oncology > General
LSN:	1-4020-0522-9
Barcode:	9781402005220

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