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Replacement of Renal Function by Dialysis (Paperback, 4th ed. 1996. Softcover reprint of the original 4th ed. 1996)
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Replacement of Renal Function by Dialysis (Paperback, 4th ed. 1996. Softcover reprint of the original 4th ed. 1996)
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In this chapter, the term 'renal osteodystrophy' will be used in a
generic sense to include all the clinical syn- dromes of skeletal
disease and altered calcium (Ca) and phosphorus (P) homeostasis
resulting from chronic renal failure (CRF). The skeletal pathology
can include osteitis TAHQgT 0BQAN8 fibrosa and other features of
secondary hyperparathy- OUT roidism, osteomalacia, aplastic or
adynamic bone disease, PARATHVnOO QLAND osteoporosis,
osteosclerosis, and in children, retardation of growth. Emphasis
will be placed on the manifestations Figure I. Schema showing the
conversion of vitamin D3 (D3) of these syndromes that occur in
patients on maintenance to 25-hydroxyvitamin D3 (25(OH)D3 or
calciferol) in the liver dialysis with a consideration of
pathogenesis, prevention and subsequently to 1,25 dihydroxyvitamin
Dj (l,25[OH)2D3 and management. or calcitriol) in the kidney.
Vitamin D through a specific action on collagen metabolisnh. There
The term, vitamin D, is used to include vitamin D, or are also
cellular receptors for calcitriol in the parathyroid
cholecalciferol, the naturally occurring sterol present in glands,
pancreas, kidneys, and salivary glands, although animals, and
vitamin D, or ergocalciferol, a sterol gener- its action in these
tissues are uncertain. Recently, calcitriol ated through the
ultraviolet irradiation of plant precursors.
General
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