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Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic Syndrome (Hardcover, 1st ed. 2016)
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Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic Syndrome (Hardcover, 1st ed. 2016)
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This comprehensive book reviews our current state of knowledge
about the pathogenesis of idiopathic nephrotic syndrome (INS),
which comprises a heterogeneous group of diseases with distinct
histological characteristics, such as minimal-change nephrotic
syndrome (MCNS), focal segmental glomerulosclerosis (FSGS), and
idiopathic membranous nephropathy (IMN). As the word "idiopathic"
indicates, the pathogenesis of INS remains unclear. Historically,
T-cell dysfunction has been thought to play an important part in
the pathogenesis of MCNS, while circulating vascular permeabilities
have been believed to induce proteinuria in FSGS. The book further
describes recent advances in molecular biology, which have allowed
us to speculate on the interactions between visceral glomerular
epithelial cells (podocytes) and the relative significance of
several molecules in the pathogenesis of INS, such as reactive
oxygen species, nuclear factor-kappa B, CD80, angiopoietin-like 4,
cardiotrophin-like cytokine-1, and M-type phospholipase A2
receptor. The normally rapid pace of scientific progress
occasionally devolves into a state of chaos, and the pathogenetic
research on INS is one such case. This volume will help researchers
and scientists to collaborate, share resources, and expedite the
design of protocols to evaluate the putative factors.
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