Books > Medicine > Clinical & internal medicine > Musculoskeletal medicine
|
Buy Now
The Plasticity of Skeletal Muscle - From Molecular Mechanism to Clinical Applications (Hardcover, 1st ed. 2017)
Loot Price: R5,711
Discovery Miles 57 110
|
|
The Plasticity of Skeletal Muscle - From Molecular Mechanism to Clinical Applications (Hardcover, 1st ed. 2017)
Expected to ship within 12 - 17 working days
|
This book discusses recent advances and various topics in
plasticity of skeletal muscle from the perspectives of morphology,
biological function, and clinical applications. Skeletal muscle is
a highly plastic organ to adapt to environmental various demands,
appears to endocrine various myokines, which flow into blood to
protect the recognizing function of brain and inhibit the
appearance of several cancer tumorigenesis. The book deals with
current stem-cell based, pharmacological, and nutritional therapies
for muscle wasting (sarcopenia, cachexia, and muscular dystrophy).
It also explains the roles of biological mediators such as PGC-1,
transient receptor potential cation channels (TRPC), and AMPK in
modulating muscle function. The functional roles of
ubiquitin-proteasome system, autophagy-dependent signaling in
muscle homeostasis, ribosome biogenesis, and redox regulation of
mechanotransduction to modulate skeletal muscle mass are also
covered. It is an essential resource for physicians, researchers,
post-docs as well as graduate students in the field of sports
science including rehabilitation therapy, exercise physiology,
exercise biochemistry, and molecular biology dealing with skeletal
muscle.
General
Is the information for this product incomplete, wrong or inappropriate?
Let us know about it.
Does this product have an incorrect or missing image?
Send us a new image.
Is this product missing categories?
Add more categories.
Review This Product
No reviews yet - be the first to create one!
|
|
Email address subscribed successfully.
A activation email has been sent to you.
Please click the link in that email to activate your subscription.