Alternative splicing occurs in most human genes and contributes to
protein diversity by producing multiple mRNAs from each gene. In
this book, the authors present new developments in alternative
splicing research. Topics discussed include alternative splicing
alterations in Alzheimer's disease; plant RNA-binding proteins
implicate mRNA processing in abscisic acid (ABA) responses;
comprehensive analyses of alternative exons in neuronally
differentiated P19 cells; an epigenetic view on alternative
splicing; identification of genuine alternative splicing variants
for rare or long-sized transcripts; alternative RNA splicing and
regulation of nitric oxide signalling; alternative splicing by
analysing human mRNA diversity using data of FLJ human cDNAs;
alternative splicing in human immune systems and auto-immune
diseases; and poly (ADP-Ribosyl)ation regulation in alternative
splicing.
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