In industrialized countries, ischemic heart disease is by far the
most common organ-specific cause of death. The thrombotic occlusion
of a coronary artery which had previously been severely altered by
atherosclerosis, is the most frequent cause of ischemic
deterioration of myocardial tissue, i. e. myocardial infarction.
Death of the human individual occurs when myocardial ischemia
causes a critical impairment of cardiac pump function. The failure
of a heart with an ischemic area may be due to the amount and
location of contractile tissue becoming paralyzed or even necrotic,
or to arrhythmias provoked by the ischemic condition, or by a
combination of both factors. Considerable progress has been made in
the development of antiarrythmic therapy. Effective tools have been
developed to reperfuse ischemic myocar dial tissue as soon as the
patient reaches hospital. However, therapeutical principles for the
ischemic-reperfused myocardium which would specifically interfere
with the state of injury of the ischemic tissue at the onset of
reperfusion, and avoid the apparent hazards of the reperfusion
process itself, have yet to be established. But not only approved
therapeutical concepts are lacking, the pathophysiology of
myocardial cell injury in pro gressive ischemia and under
reperfusion is in itself only partly understood.
General
| Imprint: |
Springer
|
| Country of origin: |
Netherlands |
| Series: |
Developments in Cardiovascular Medicine, 104 |
| Release date: |
March 1990 |
| First published: |
1990 |
| Editors: |
H.M. Piper
|
| Dimensions: |
250mm (L) |
| Format: |
Hardcover
|
| Pages: |
440 |
| Edition: |
1990 ed. |
| ISBN-13: |
978-0-7923-0459-3 |
| Categories: |
Books
Promotions
|
| LSN: |
0-7923-0459-4 |
| Barcode: |
9780792304593 |
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