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1. Renal failure following circulatory shock develops because of
per- sistent vasoconstriction which is just sufficient to prevent
glomerular filtration. Hypoxia of renal tissue has not been
demonstrated in surviving cases. 2. During the low-pressure phase
in circulatory shock the remaining blood flow through the medullary
regions washes out the osmotic gradient built up by the
countercurrent system of Henle's loops, so that a concentrated
urine cannot be formed. 3. During recovery from circulatory failure
the osmotic gradient of the medullary region can only be built up
if sufficient fluid from the glomerular filtrate reaches the
countercurrent system. The greater the GF, the faster the gradient
is built up. References 1. BoYLAN, J. W. , and E. AssHAUER:
Unpublished data. -2. DEETJEN, P. , and K. KRAMER: Pfliigers Arch.
Physiol. (G. ) (in press). - 3. KRAMER, K. , and K. ULLRICH:
Pfliigers Arch. Physiol. (G. ) 267,251 (1958). -4. KRA- MER, K. ,
K. THURAU and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 270, 251
(1960). - 5. KRAMER, K. , and P. DEETJEN: Pfliigers Arch. Physiol.
(G. ) 271, 782 (1960). -6. KRAMER, K. , and P. DEETJEN: Unpublished
data. - 7. KuHN, W. , and A. RAMEL: Helv. chim. acta 42,628 (1959).
- 8. LASSEN, N. A. , 0. MuNcK and J. H. THAYSEN: Acta physiol.
Scand. IJ1, 371 (1961). - 9. MUNCK, 0. : Renal Circulation in Acute
Renal Failure. Oxford 1958.
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