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The factors affecting blood vitamin C levels are described in
detail in this series. Many factors such as aging, smoking,
infection, trauma, surgery, hemolysis, hormone administration,
heavy metals, pregnancy, alcohol, ionizing radiation and several
medicines have been found to cause a disturbance of ascorbic acid
metabolism and to reduce blood vitamin C levels. Indeed,
abnormalities of ascorbic acid metabolism, due to factors such as
smoking, occur much more frequently than does dietary vitamin C
deficiency today.It is now known that low blood vitamin C levels
are associated with histaminemia (high blood histamine levels), and
also that ascorbate-responsive histaminemia is common in apparently
healthy people. High blood histamine levels are believed to cause
small hemorrhages within the inner walls of the blood vessels and
these may lead to the deposition of cholesterol, as an aberrant
form of wound healing. Ascorbic acid not only reduces blood
histamine levels, but also aids the conversion of cholesterol to
bile acids in the liver. The clinical pathological and chemical
changes observed in ascorbic acid deficiency are discussed in
detail. Several diseases and disorders associated with low blood
vitamin C levels are also described. Possible toxic effects
resulting from the oxidation of ascorbic acid are noted, and
reasons for the use of D-catechin or other chelating fiber to
prevent or minimize the release of ascorbate-free radical are
detailed. An excellent reference for physicians, nutritionists and
other scientists
The factors affecting blood vitamin C levels are described in
detail in this series. Many factors such as aging, smoking,
infection, trauma, surgery, hemolysis, hormone administration,
heavy metals, pregnancy, alcohol, ionizing radiation and several
medicines have been found to cause a disturbance of ascorbic acid
metabolism and to reduce blood vitamin C levels. Indeed,
abnormalities of ascorbic acid metabolism, due to factors such as
smoking, occur much more frequently than does dietary vitamin C
deficiency today.It is now known that low blood vitamin C levels
are associated with histaminemia (high blood histamine levels), and
also that ascorbate-responsive histaminemia is common in apparently
healthy people. High blood histamine levels are believed to cause
small hemorrhages within the inner walls of the blood vessels and
these may lead to the deposition of cholesterol, as an aberrant
form of wound healing. Ascorbic acid not only reduces blood
histamine levels, but also aids the conversion of cholesterol to
bile acids in the liver. The clinical pathological and chemical
changes observed in ascorbic acid deficiency are discussed in
detail. Several diseases and disorders associated with low blood
vitamin C levels are also described. Possible toxic effects
resulting from the oxidation of ascorbic acid are noted, and
reasons for the use of D-catechin or other chelating fiber to
prevent or minimize the release of ascorbate-free radical are
detailed. An excellent reference for physicians, nutritionists and
other scientists
The factors affecting blood vitamin C levels are described in
detail in this series. Many factors such as aging, smoking,
infection, trauma, surgery, hemolysis, hormone administration,
heavy metals, pregnancy, alcohol, ionizing radiation and several
medicines have been found to cause a disturbance of ascorbic acid
metabolism and to reduce blood vitamin C levels. Indeed,
abnormalities of ascorbic acid metabolism, due to factors such as
smoking, occur much more frequently than does dietary vitamin C
deficiency today.It is now known that low blood vitamin C levels
are associated with histaminemia (high blood histamine levels), and
also that ascorbate-responsive histaminemia is common in apparently
healthy people. High blood histamine levels are believed to cause
small hemorrhages within the inner walls of the blood vessels and
these may lead to the deposition of cholesterol, as an aberrant
form of wound healing. Ascorbic acid not only reduces blood
histamine levels, but also aids the conversion of cholesterol to
bile acids in the liver. The clinical pathological and chemical
changes observed in ascorbic acid deficiency are discussed in
detail. Several diseases and disorders associated with low blood
vitamin C levels are also described. Possible toxic effects
resulting from the oxidation of ascorbic acid are noted, and
reasons for the use of D-catechin or other chelating fiber to
prevent or minimize the release of ascorbate-free radical are
detailed. An excellent reference for physicians, nutritionists and
other scientists
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