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Abnormal expression of MHC class I molecules in malignant cells is
a frequent occurrence that ranges from total loss of all class I
antigens to partial loss of MHC specific haplotypes or alleles.
Different mechanisms are described to be responsible for these
alterations, requiring different therapeutic approaches. A complete
characterization of these molecular defects is important for
improvement of the strategies for the selection and follow-up of
patients undergoing T-cell based cancer immunotherapy. Precise
identification of the mechanism leading to MHC class I defects will
help to develop new personalized patient-tailored treatment
protocols. There is significant new research on the prevalence of
various patterns of MHC class I defects and the underlying
molecular mechanisms in different types of cancer. In contrast, few
data is available on the changes in MHC class I expression during
the course of cancer immunotherapy, but the authors have recently
made discoveries that show the progression or regression of a tumor
lesion in cancer patients undergoing immunotherapy depends on the
molecular mechanism responsible for the MHC class I alteration and
not on the type of immunotherapy used. According to this notion,
the nature of the preexisting MHC class I lesion in the cancer cell
has a crucial impact on determining the final outcome of cancer
immunotherapy. This SpringerBrief will present how MHC class 1 is
expressed, explain its role in tumor progression, and its role in
resistance to immunotherapy.
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