It is vital to have knowledge of the neuroanatomical structures and functional neurological mechanisms, which are disrupted in neurogenic speech/language, disordered persons in order to understand the speech/language deficits themselves.

This book provides a comprehensive coverage of the neurological basis of both the clinically recognised forms of aphasia and the various motor speech disorders, in both children and adults. It also covers more recently recognised language disorders, such as Parkinsons and related diseases, right hemisphere damage, closed-head injury, dementia, etc. This is a perfect text for practitioners who need to understand the integration of neuroanatomy and functional neurology with the practice of speech-language pathology.

This book is the first to provide comprehensive coverage of the communication impairments that occur in association with multiple sclerosis. It covers not only the more widely recognised motor speech disorders seen in association with this condition, but also the more recently identified language disorders reported to occur in persons with diseases of the cerebral white matter. A full description of the neuropathophysiology, epidemiology, aetiology, clinical manifestations and medical treatment of multiple sclerosis is also included.

This book provides comprehensive coverage of speech and language disorders arising from pathological processes involving the subcortical structures of the brain. It gives an understanding of these disorders in terms of their neuropathological basis, clinical symptomatology and prognosis. A full discussion of contemporary models and theories of subcortical participation in speech and language processing is given, including discussion of the possible roles of structures such as the basal ganglia, subthalamic nucleus, thalamus and cerebellum.

The book covers speech and language disorders associated with a variety of subcortical conditions, ranging from major degenerative conditions such as Parkinsons' Disease, Huntington's chorea and dystonia, through to acquired non-degenerative subcortical lesions arising from, for example, cerebrovascular accidents and sterotactic surgically induced lesions. In addition, a full description of the relevant assessment and treatment procedures currently recommended for use for each of the subcortical communication disorders is given.