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As in so many fields of scientific endeavour following the
molecular biology revo lution, our knowledge of the role of
radicals not only in pathological states, but in basic physiology
has developed exponentially. Indeed, our evolving concepts have,
like so many political parties, been forced into dramatic "V-turns"
and contortions. Within our working lives, we have had to debate
whether radicals made any con tribution to any pathology, whilst
now it is difficult not to entertain the view that every
physiological process is pivotally controlled by exquisitely
sensitive radical reactions. Inflammation is, of course, an example
of pathology evolving from physiology, and in this book we have
called upon both scientists and clinicians who have research
interests in the complex switching mechanisms that sustain these
transi tions. The book as a whole explores, from a physiological
standpoint, how deter ministic radical systems sensitive to their
initial conditions can interdigitate, iterate and feed back to
control diverse cellular processes that create the inflammatory
response. Whilst systems such as these to a mathematician would
provide the basis for a chaotic response, one is forced to marvel
how, for all stages of an inflammatory reaction, this system
appears exquisitely controlled, making therapeutic manipula tion
both possible and, to some extent, predictable."
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