Chronic diseases have rapidly become the leading global cause of morbidity and mortality, yet there is poor understanding of this transition, or why particular social and ethnic groups are especially susceptible. In this book, Wells adopts a multidisciplinary approach to human nutrition, emphasising how power relations shape the physiological pathways to obesity, diabetes, hypertension and cardiovascular disease. Part I reviews the physiological basis of chronic diseases, presenting a 'capacity-load' model that integrates the nutritional contributions of developmental experience and adult lifestyle. Part II presents an evolutionary perspective on the sensitivity of human metabolism to ecological stresses, highlighting how social hierarchy impacts metabolism on an intergenerational timescale. Part III reviews how nutrition has changed over time, as societies evolved and coalesced towards a single global economic system. Part IV integrates these physiological, evolutionary and politico-economic perspectives in a unifying framework, to deepen our understanding of the societal basis of metabolic ill-health.

This comprehensive synthesis of current medical and evolutionary literature addresses key questions about the role body fat plays in human biology. It explores how body energy stores are regulated, how they develop over the life-course, what biological functions they serve, and how they may have evolved. There is now substantial evidence that human adiposity is not merely a buffer against the threat of starvation, but is also a resource for meeting the energy costs of growth, reproduction and immune function. As such it may be considered as important in our species evolution as other traits such as bipedalism, large brains, and long life spans and developmental periods. Indeed, adiposity is integrally linked with these other traits, and with our capacity to colonise and inhabit diverse ecosystems. It is because human metabolism is so sensitive to environmental cues that manipulative economic forces are now generating the current obesity epidemic.

Chronic diseases have rapidly become the leading global cause of morbidity and mortality, yet there is poor understanding of this transition, or why particular social and ethnic groups are especially susceptible. In this book, Wells adopts a multidisciplinary approach to human nutrition, emphasising how power relations shape the physiological pathways to obesity, diabetes, hypertension and cardiovascular disease. Part I reviews the physiological basis of chronic diseases, presenting a 'capacity-load' model that integrates the nutritional contributions of developmental experience and adult lifestyle. Part II presents an evolutionary perspective on the sensitivity of human metabolism to ecological stresses, highlighting how social hierarchy impacts metabolism on an intergenerational timescale. Part III reviews how nutrition has changed over time, as societies evolved and coalesced towards a single global economic system. Part IV integrates these physiological, evolutionary and politico-economic perspectives in a unifying framework, to deepen our understanding of the societal basis of metabolic ill-health.

This comprehensive synthesis of current medical and evolutionary literature addresses key questions about the role body fat plays in human biology. It explores how body energy stores are regulated, how they develop over the life-course, what biological functions they serve, and how they may have evolved. There is now substantial evidence that human adiposity is not merely a buffer against the threat of starvation, but is also a resource for meeting the energy costs of growth, reproduction and immune function. As such it may be considered as important in our species evolution as other traits such as bipedalism, large brains, and long life spans and developmental periods. Indeed, adiposity is integrally linked with these other traits, and with our capacity to colonise and inhabit diverse ecosystems. It is because human metabolism is so sensitive to environmental cues that manipulative economic forces are now generating the current obesity epidemic.

Recent research has emphasized that socially transmitted information may affect both the gene pool and the phenotypes of individuals and populations, and that an improved understanding of evolutionary issues is beneficial to those working towards the improvement of human health. Equally, an improved awareness of how human behavior influences health and reproductive fitness is starting to shed new light on the processes that shape the evolution of human behavior and the human mind.

Focusing directly on these emerging trends, Social Information Transmission and Human Biology bridges the gap between primarily theoretical work undertaken by those with evolutionary interests and biomedical work undertaken by those dealing with practical issues in human health and demographics. Incorporating papers from a symposium organized under the auspices of the UK Society for the Study of Human Biology, this volume merges the perspectives of internationally renowned evolutionary and theoretical biologists, zoologists, anthropologists, archaeologists, psychologists, and medical researchers whose work is linked by common themes addressing how information is transmitted socially and how its transmission influences both immediate and evolutionary biological outcomes. Emphasizes the diverse ways in which socially transmitted information impacts on human biology

To illustrate these themes, the chapters draw on models and data ranging from observations on chimpanzee populations in the wild and on the human archaeological record, to studies of contemporary humans in both developing and industrialized countries. Taking a broad approach, many of the chapters address areas of behavior that are familiar toscientists in particular fields, but do so using a variety of cross-disciplinary perspectives, which will prove stimulating for researchers in a range of academic subject areas, while helping to facilitate closer collaboration between biological and social scientists.