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This book highlights the multifaceted roles of Reactive Oxygen
Species (ROS) in modulating normal cellular and molecular
mechanisms during the development of different types of heart
disease. Each chapter in the book deals with the role that altered
redox homeostasis plays in the pathophysiology of heart disease. In
addition, the book explains how reactive oxidant species interact
with their targets and provides novel strategies for attenuating
oxidative stress-induced types of heart disease. The book not only
covers ROS-induced response in heart disease at the cellular level,
but also demonstrates that an imbalance of redox states has its
roots in our genes, and explains the ways gene expression is
regulated. In turn, it reviews potential sources of ROS, their
pathological effects on the heart, and potential sites for
therapeutic interventions.
This book highlights the multifaceted roles of Reactive Oxygen
Species (ROS) in modulating normal cellular and molecular
mechanisms during the development of different types of heart
disease. Each chapter in the book deals with the role that altered
redox homeostasis plays in the pathophysiology of heart disease. In
addition, the book explains how reactive oxidant species interact
with their targets and provides novel strategies for attenuating
oxidative stress-induced types of heart disease. The book not only
covers ROS-induced response in heart disease at the cellular level,
but also demonstrates that an imbalance of redox states has its
roots in our genes, and explains the ways gene expression is
regulated. In turn, it reviews potential sources of ROS, their
pathological effects on the heart, and potential sites for
therapeutic interventions.
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