Abraham argues that a theological imagination can expand the contours of postcolonial theory through a reexamination of notions of subjectivity, gender, and violence in a dialogical model with Karl Rahner. She questions of whether postcolonial theory, with its disavowal of religious agency, can provide an invigorating occasion for Catholic theology.

Prostaglandins, Leukotrienes, and Cancer is a multi-volume series which focuses on an emerging area of cancer research. In 1968, R. H. Williams first reported that elevated prostaglandin levels are present in human medullary carcinoma. Since that time, the concept that arachidonic acid metabolites may be involved in cancer has expanded to include every aspect of the disease from cell transformation through metastasis. Prostaglandins and leukotrienes are generic terms used to describe a family ofbioactive lipids produced from unsaturated fatty acids (principally from arachidonic acid) via the cyclooxygenase and lipoxygenase pathways, respectively. Cyclooxygenase products consist of diverse products such as prostaglandin E2 (PGE2), prostacyclin (PGI2) and thromboxane A2 (TXA2), whereas lipoxygenase products consist ofhydroperoxy fatty acids and mono-, di-and tri-hydroxy acids including leukotrienes, lipoxins, and epoxides. The precursor fatty acids for the cyclooxygenase and lipoxygenase pathways are present in cellular phospholipids. This finding established an important control point in their biosynthesis --the release of substrate. This occurs in response to numerous stimuli that act at the cell surface. Dr. Bengt Samuelsson's extensive study ofthe metabolism of prostaglandins indicate that they are rapidly inactivated on a single pass through pulmonary circulation. Thus, they cannot act as circulating hormones and appear to be made on demand in the vicinity oftarget tissues leading to the concept that prostaglandins are local rrormones or autocoids. Altered production, qualitative and/or quantitative, of prostaglandins and leukotrienes has been implicated in the development of a number of disease states (e. g.

Not sure how to interpret the wealth of data in front of you?

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The benefits of this book include: An overview of the normal ranges of test results, followed by a consideration of the differential diagnoses suggested by variance from these valuesArranged by system to allow quick access to the key investigations encountered in different specialtiesA summary 'patient data' chapter to bring the different specialties together, providing an overview to completing investigation documentation and chartsSummary table and bullet point format, with a full index, to aid rapid retrieval of informationEach chapter reviewed by a specialist to ensure an accurate, practical approach to data interpretation

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Prostaglandins, Leukotrienes, and Cancer is a multi-volume series which focuses on an emerging area of cancer research. In 1968, R. H. Williams first reported that elevated prostaglandin levels are present in human medullary carcinoma. Since that time, the concept that arachidonic acid metabolites may be involved in cancer has expanded to include every aspect of the disease from cell transformation through metastasis. Prostaglandins and leukotrienes are generic terms used to describe a family ofbioactive lipids produced from unsaturated fatty acids (principally from arachidonic acid) via the cyclooxygenase and lipoxygenase pathways, respectively. Cyclooxygenase products consist of diverse products such as prostaglandin E2 (PGE2), prostacyclin (PGI2) and thromboxane A2 (TXA2), whereas lipoxygenase products consist ofhydroperoxy fatty acids and mono-, di-and tri-hydroxy acids including leukotrienes, lipoxins, and epoxides. The precursor fatty acids for the cyclooxygenase and lipoxygenase pathways are present in cellular phospholipids. This finding established an important control point in their biosynthesis --the release of substrate. This occurs in response to numerous stimuli that act at the cell surface. Dr. Bengt Samuelsson's extensive study ofthe metabolism of prostaglandins indicate that they are rapidly inactivated on a single pass through pulmonary circulation. Thus, they cannot act as circulating hormones and appear to be made on demand in the vicinity oftarget tissues leading to the concept that prostaglandins are local rrormones or autocoids. Altered production, qualitative and/or quantitative, of prostaglandins and leukotrienes has been implicated in the development of a number of disease states (e. g.

Links theory and practice for investment professionals and portfolio managers, demonstrating why some portfolios consistently perform better than others Investing well, like any other business, depends on competitive advantage. Some portfolios reliably generate greater returns than others because they simply are better positioned to benefit from strengths and avoid weaknesses. Building and using competitive advantage becomes central to the daily work of the best mutual funds, hedge funds, banks, insurers and virtually every other type of portfolio. But competitive advantage commonly is overlooked in most written work for investment professionals. The literature often varies between abstract formal treatments and pragmatic workbooks with little in between. Competitive Advantage in Investing fills the gap by integrating modern portfolio theory with actual practice in one comprehensive volume. This innovative book guides investment professionals on building and sustaining competitive advantage and helps policymakers and researchers apply theory in a wide range of practical settings. Author Steven Abrahams--Senior Managing Director at Amherst Pierpont Securities and former Adjunct Professor of Finance and Economics at Columbia Business School--draws from his experience in both academic theory and real-life strategic investing to bridge the two worlds. This valuable resource: Connects the formal literature on investing to the actual work of most institutional portfolio managers Examines core strengths and weaknesses that drive portfolio behavior at mutual and hedge funds, banks and insurers, at other institutions and for individuals Demonstrates how linking portfolio theory and practice can increase competitive advantage Offers a robust description of investing, markets, and asset value Competitive Advantage in Investing: Building Winning Professional Portfolios is a must-have book for any investment professional, policymaker, or researcher.

Abraham argues that a theological imagination can expand the contours of postcolonial theory through a reexamination of notions of subjectivity, gender, and violence in a dialogical model with Karl Rahner. She questions of whether postcolonial theory, with its disavowal of religious agency, can provide an invigorating occasion for Catholic theology.

Tort Law and the Construction of Change studies the interaction of law and social change in American history. Tort law-civil law made by judges, not legislators-is traditionally thought to arise out of legal precedent. But Kenneth S. Abraham and G. Edward White show that American judges over the course of the previous two centuries also paid close attention to changing societal contexts in which lawsuits for civil injuries arose. They argue that two versions of history-one grounded in the application of previous legal rules and the other responsive to larger societal changes-must be considered in tandem to grasp fully how American civil law has evolved over time. In five fascinating chapters, they cover understudied areas of tort law, such as liability for nonphysical harm-including lawsuits for defamation, privacy, emotional distress, sexual harassment, and the hacking of confidential information-and aspects of tort litigation that have now disappeared, such as the prohibition against ""interested"" parties testifying in civil actions or the intentional infliction of temporal damage without justification. What emerges is a picture of the complicated legal dance American judges performed to cloak their decisions to make at times radical changes in tort law in response to social transformations. When confronting established tort doctrines under pressure from emerging social changes, they found ways to preserve at least the appearance of doctrinal continuity.

This paper demonstrates how a better understanding of culture can lend itself to a better understanding of meaning in discourse between two cultures who operate across a linguistic barrier. The paper falls under the tradition of cultural relativism, and the modern version of linguistic relativity. Culture is a multi-faceted concept that can mean something different depending on the objective of study, and the particular discipline of the researcher. This led the researcher to adopt a theory of culture as socially distributed knowledge. Due to the particular subject related to meaning and linguistic contact, the paper makes language the parameter to define the culture being studied, which in this case is the Arabic speech community, writ large. Obviously, there are many subordinate cultures in such a large entity. The particular theory and parameter that we adopted will allow us to make some generalizations about Arabic speakers, based on a study of characteristics of the Arabic language. The theory also allows for human indeterminism, which tells us that each individual human being acts on his own accord, sometimes in accordance with cultural norms and sometimes not. This helps to keep the project in perspective and to prevent rash action based on expectations of behavior that can never be accurately predicted. About one third of this paper is dedicated to gaining an understanding of the history and characteristics of the Arabic language varieties. Because the Arabic speech community is the culture being studied, and language reflects the particular history and traditions of those who speak it, this amount of space is justified. In this case, the roots of Arabic language diglossia, and the different ways that the language has become a symbol of nationalism, pan-Arabism, and pan-Islamic ideology are of interest to the study and are the focus for the history. There is no foray into the realm of linguistics as a science, nor the large corpus of Arabic language literat

Kenneth Abraham explores the development and interdependency of the tort liability regime and the insurance system in the United States during the twentieth century and beyond, including the events of September 11, 2001.

From its beginning late in the nineteenth century, the availability of liability insurance led to the creation of new forms of liability, heavily influenced expansion of the liabilities that already existed, and continually promoted increases in the amount of money that was awarded in tort suits. A "liability-and-insurance spiral" emerged, in which the availability of liability insurance encouraged the imposition of more liability, and, in turn, the imposition of liability encouraged the further spread of insurance.

Liability insurance was not merely a source of funding for ever-greater amounts of tort liability. Liability insurers came to dominate tort litigation. They defended lawsuits against their policyholders, and they decided which cases to settle, fight, or appeal. The very idea behind insurance--that spreading losses among large numbers of policyholders is desirable--came to influence the ideology of tort law. To serve the aim of loss spreading, liability had to expand.

Today the tort liability and insurance systems constantly interact, and to reform one the role of the other must be fully understood.