Rheumatoid arthritis (RA) is the most common and most severe form
of inflammatory arthritis. The pathogenesis of RA has been the
subject of intense research for several decades. The prevailing
hypotheses have changed over the years, and have attempted to
incorporate the most recent data. Although T cells represent an
important component of the cells which infiltrate the joint
synovium, their contribution at a late stage of the disease remains
a matter of debate. The goal of this book is to outline the major
arguments and data suggesting that T cells may, or may not, be
central players in the pathogenesis of chronic RA. While each of
the editors and authors has his/her own bias (as will be clear by
reading the respective chapters), our hope is that the readers will
enjoy a complete and balanced view of the critical questions and
experiments. This is not just an intellectual exercise since the
direction of future therapeutic interventions depends heavily on
how one interprets the pathogenesis of RA and the contribution of T
cells.
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