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What we now call 'deep venous thrombosis' (DVT) has been
elucidated by a diversity of investigative approaches during the
past four centuries. The authors of this book survey the history of
the field and ask: why has one of these perspectives - the
haematological/biochemical - come to dominate research into the
causation of DVT during the past 50 years and to exclude
alternatives? In answering this question, the authors show that the
current consensus model is conceptually flawed.
What we now call a deep venous thrombosisa (TM) (DVT) has been
elucidated by a diversity of investigative approaches during the
past four centuries. The authors of this book survey the history of
the field and ask: why has one of these perspectives a the
haematological/biochemical a come to dominate research into the
causation of DVT during the past 50 years and to exclude
alternatives? In answering this question, they show that the
current consensus model is conceptually flawed. Building on the
work of William Harvey, John Hunter, Rudolf Virchow, Ludwig Aschoff
and a number of pathologists in the mid-20th century, they offer a
revised account of the aetiology of this condition. In the process
they retrace and review the 160-year-old philosophical and
methodological schism in biomedical research and, using DVT as an
example, propose how this schism might be bridged to the benefit of
both research and clinical practice.
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