What we now call 'deep venous thrombosis' (DVT) has been
elucidated by a diversity of investigative approaches during the
past four centuries. The authors of this book survey the history of
the field and ask: why has one of these perspectives - the
haematological/biochemical - come to dominate research into the
causation of DVT during the past 50 years and to exclude
alternatives? In answering this question, the authors show that the
current consensus model is conceptually flawed.
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