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The POW Who Saved Kashmir - Unsung Saga of Sher Bachha Brig Pritam Singh, MC (Paperback): Pankaj P. Singh, Jasbir Singh Sm The POW Who Saved Kashmir - Unsung Saga of Sher Bachha Brig Pritam Singh, MC (Paperback)
Pankaj P. Singh, Jasbir Singh Sm
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R257 Discovery Miles 2 570 Ships in 10 - 15 working days
Long Trek to India and Freedom - Daring Escape by Three Indian Army Officers from Japanese POW Camp (Paperback): Singh Jasbir Long Trek to India and Freedom - Daring Escape by Three Indian Army Officers from Japanese POW Camp (Paperback)
Singh Jasbir
R760 Discovery Miles 7 600 Ships in 10 - 15 working days

This is the little known story of a daring escape, by three young officers of Indian Army (IA), from a Japanese World War II Prisoner of War (POW) Camp, and their torturous trek to India and freedom. The trio escaped from POW Camp in Singapore in May 1942 and made their way through Malaya (now Malaysia), Thailand and Burma (now Myanmar), to reach India. In Burma they split into two groups. Balbir & Parab moved north through Myitkyina and enemy-held territories, before they contacted an allied patrol in the heavily jungled, hill tracts of upper Burma.

Serum Uric Acid - Cause or Consequence in Hypertension ? (Paperback): Chaudhary Vijay, Singh Jasbir, Pandey Rajesh Serum Uric Acid - Cause or Consequence in Hypertension ? (Paperback)
Chaudhary Vijay, Singh Jasbir, Pandey Rajesh
R910 Discovery Miles 9 100 Ships in 10 - 15 working days

In India, cardiovascular diseases (CVDs) are estimated to be responsible for 1.5 million deaths annually. Indeed, it is estimated that by 2020, CVDs will be the largest cause of mortality and morbidity in India. Hypertension is a major risk factor for CVDs, including stroke and myocardial infarction, and its burden is increasing disproportionately in developing countries. It is estimated that around two-thirds of those with people with hypertension worldwide were living in developing countries (639 million) in 2000, and that this would rise to three-quarters living in developing countries (1.15 billion) by 2025. Uric acid builds up when the body makes too much of it, fails to excrete it or both. It is a metabolic product resulting from purine. It remains possible that uric acid is a marker for xanthine oxidase-associated oxidants and that the latter could be driving the hypertensive response. However, the evidence suggests that uric acid is a true modifying and possibly causal factor for human primary hypertension. Hence, early management of hyperuricemia might delay the development of essential hypertension.

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