The goal of this text is to focus readers attention on three major
areas; the origin and localization of GSH in the nervous system;
the multiple effects of GSH on neural health activity; and the
potential for alterations on GSH status to lead to neurological
damage of the type observed in amyotrophic lateral sclerosis,
Parkinson's disease and other neurological disorders. The text also
touches upon the additional roles of the antoxidant GSH, including
possible neurotransmitter action, redox modulation of ionotropic
receptor function, and neuroprotection against exicitoxic actions
of glutamate.
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