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Adenosine, Cardioprotection and Its Clinical Application (Hardcover, 1997 ed.)
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Adenosine, Cardioprotection and Its Clinical Application (Hardcover, 1997 ed.)
Series: Developments in Cardiovascular Medicine, 194
Expected to ship within 12 - 17 working days
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It has been almost 15 years since the fIrst reports appeared
indicating that adenosine exerted a .protective effect in ischemic
and reperfused myocardium. Numerous experimental studies have shown
that adenosine (both exogenous and endogenous adenosine) delays the
onset of ischemic contracture, modulates myocardial metabolism
during ischemia, attenuates reversible postischemic ventricular
dysfunction (myocardial stunning), and reduces myocardial infarct
size. Initial studies on adenosine's cardioprotective effect were
based on its ability to stimulate postischemic ATP resynthesis,
increase coronary blood flow, and reduce heart rate. Although these
actions of adenosine are undoubtedly benefIcial to the
ischemic/reperfused heart, it now appears that adenosine's
cardioprotective effect may be exclusive of these properties. The
immense growth in the number of articles on adenosine
cardioprotection in the last several years has been related in
large part to the hypothesis that adenosine plays a role in
ischemic preconditioning. Ischemic preconditioning is the
phenomenon in which a brief period of ischemia (and reperfusion)
prior to a more prolonged occlusion reduces myocardial infarct
size. This form of myocardial protection has received much interest
because ischemic preconditioning has been shown to be the most
potent means of reducing infarct size in all animal models thus far
tested. In fact prior to studies implicating adenosine's role in
ischemic preconditioning, adenosine's infarct reducing effect was
not well recognized.
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