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On June 28-29, 1990 in Noordwijk, The Netherlands, the
International Association of Inflammation societies (IAIS)
organized their first symposium on Drugs in Inflammation. The
symposium was an official satellite of the Ixth International
Congress of Pharmacology (IUPHAR). This volume contains the
proceedings from this satellite meeting. The IAIS was constituted
in 1988 when representatives of the Inflammation Research
Association (IRA), European Workshop on Inflammation (EWI) and
British Inflammation Research Association (BIRAs) met at White
Haven, Pennsylvania during the Fourth International Conference of
the IRA and the group decided that improved communication between
inflammation societies could be achieved by establishment of a
society. The IAIS would encourage and foster collaboration between
the many inflammation societies throughout the world and assist in
the establishment of new societies whenever possible. The satellite
was a major success thanks to the efforts of the planning
committee: Wim van den Berg, the local organizer, Mike Bray, the
programme chairman, Rodger McMillan, Greg Harper, Mike Parnham and
Kay Brune. In addition, the chairman and presenters ensured the
scientific content of the meeting was of a very high standard. Most
importantly, the 100 participants from 15 countries made the
sessions lively and interactive. The IAIS will hopefully organize
additional symposiums in the future. Alan J. Lewis, Ph.D.
The purpose of this volume in the Progress in Inflammation Research
series is to provide the biomedical and clinical researcher with a
state-of-the-art insight in the role of cytokines in joint
inflammation and joint destruction. This is of relevance for better
understanding of key processes in diseases such as rheumatoid
arthritis (RA) and osteoarthritis (OA). Apart from the impact of
old and novel cytokines on joint tissues, the various chapters
address the issue of targeted therapy with biological response
modifiers and future interventions with carefully designed
inhibitors. Spe cial attention is given to elements of synovial
cell activation, cell-cell interaction, cytokine interplay as well
as mechanisms of cartilage destruction and bone erosion. In
addition to an outline of the role of established cytokines, such
as TNF, IL-l and IL-6, new information is given on the novel
cytokines IL-15, IL-17 and IL-18 and their positioning in the
complex cytokine interplay. Cytokine regulation of destructive
enzymes, RANKL, the endogenous inhibitor OPG and their crucial
roles as central players in joint erosion are highlighted.
Together, the chapters provide a complete and balanced view on
pivotal cytokines and joint pathology.
The purpose of this volume in the Progress in Inflammation Research
series is to provide the biomedical and clinical researcher with a
state-of-the-art insight in the role of cytokines in joint
inflammation and joint destruction. This is of relevance for better
understanding of key processes in diseases such as rheumatoid
arthritis (RA) and osteoarthritis (OA). Apart from the impact of
old and novel cytokines on joint tissues, the various chapters
address the issue of targeted therapy with biological response
modifiers and future interventions with carefully designed
inhibitors. Spe cial attention is given to elements of synovial
cell activation, cell-cell interaction, cytokine interplay as well
as mechanisms of cartilage destruction and bone erosion. In
addition to an outline of the role of established cytokines, such
as TNF, IL-l and IL-6, new information is given on the novel
cytokines IL-15, IL-17 and IL-18 and their positioning in the
complex cytokine interplay. Cytokine regulation of destructive
enzymes, RANKL, the endogenous inhibitor OPG and their crucial
roles as central players in joint erosion are highlighted.
Together, the chapters provide a complete and balanced view on
pivotal cytokines and joint pathology.
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