Quinolones are among the most commonly prescribed antimicrobials
because of their broad-spectrum antimicrobial activity, which
inhibit DNA synthesis by targeting two related but functionally
distinct and essential type II topoisomerase, DNA gyrase and
topoisomerase IV. The quinolone resistance is traditionally
mediated by the mutation of chromosomal genes encoding DNA gyrase
and/or topoisomerase IV. Plasmid-mediated resistance mechanisms
also play a significant role in quinolone resistance. In recent
years, a bacterial resistance against the quinolones has increased
significantly, and no previous Iraqi survey has evaluated clinical
isolates of Escherichia coli with reduce susceptibility to
quinolones. This book provides a new data on the occurrence and
diversity of quinolones resistance genes in E. coli strains
isolated from patients with urinary tract infection in Iraqi
hospitals.
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