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Hormonal Carcinogenesis III - Proceedings of the Third International Symposium (Paperback, Softcover reprint of the original 1st ed. 2001)
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Hormonal Carcinogenesis III - Proceedings of the Third International Symposium (Paperback, Softcover reprint of the original 1st ed. 2001)
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Since our previous symposium in 1995, the pace of research in
hormones and cancer has accelerated. Progress in our understanding
of hormonal carcinogenic processes has been a direct result of the
advances made in cell biology, endocrinology, and carcinogenesis at
the molecular level. The newer fields of molecular genetics and
cytogenetics already have and are expected to continue to playa
major role in furthering our understanding of the cellular and
molecular events in hormonal carcinogenesis. It has become
increasingly clear that the risk of naturally occurring sex
hormones in carcinogenic processes, both in human and in animal
models, requires only minute quantities of hormones, at both the
serum and tissue levels. Moreover, hormone target tissues for
neoplastic transformation, perhaps with the exception of the liver,
generally have relatively modest ability to metabolize sex
hormones, such as the breast and prostate. Table 1 summarizes the
serum, and in most cases, the tissue levels of sex hormones, both
endogenously and exogenously ingested, which are associated with
increased risk for endocrine-associated cancers such as breast,
endometrium, and prostate, as well as the hormone levels of four
experimental models that have been shown to elicit high tumor
incidences. In contrast to the human, in which the hormone levels
are cyclic, however, the latter require continuous hormone exposure
at these relatively low levels.
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