Stroke is a major cause of death and disability in the U.S. and
worldwide. A variety of pathophysiologic episodes or cellular
medications occur following a stroke, and knowledge of these
aftermath events can lead to potential therapeutic strategies that
may reverse or attenuate stroke injury. Cellular events that occur
following stroke include the excessive releases of excitatory amino
acids, alterations in the genomic responses, mitochondrial injury
producing reactive oxygen and nitrogen species (ROS), and secondary
injury, often in the setting of reperfusion.
General
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