Fresh insights into the pathogenic mechanisms by which
hyperglycemia induces tissue and organ injurt are the basis for
rapidly evolving promising therapies in diabetes. Especially
promising as targets for intervention are products of oxidative
stress, including kinins and growth factors. Improving results of
renal replacement regimes now incorporating pancreatic islet
transplants are able to delay and prevent end-organ damage in
diabetic individuals. The evolving story of the taming of diabetes
is of direct concern to nephrologists, endocrinologists,
ophthalmologists, primary care physicians and medical students.
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