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Homocysteine Metabolism: From Basic Science to Clinical Medicine (Hardcover, 1997 ed.)
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Homocysteine Metabolism: From Basic Science to Clinical Medicine (Hardcover, 1997 ed.)
Series: Developments in Cardiovascular Medicine, 196
Expected to ship within 10 - 15 working days
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In 1962, 30 years after the discovery by du Vigneaud have
pathologic consequences. One potentially sig- of a new sulfur amino
acid, homocysteine; Carson and nificant health outcome of such mild
to moderate Neil reported two siblings with mental retardation in
hyperhomocysteinemia is an increased risk of occlu- northern
Ireland with elevated urinary homocystine. sive vascular disease.
Homocysteine concentrations in Nearly simultaneously, Gerritsen and
Waisman patients with vascular disease were, on average, 31 %
greater than in normal controls. Prospective assess- identified
increased homocystine in the urine of a mentally retarded infant in
Wisconsin. Within two ment of vascular disease risk among men with
higher years, Harvey Mudd, James Finkelstein, and their
homocysteine concentrations indicated that plasma coworkers at the
National Institutes of health (USA) homocysteine at only 12% above
the upper limit of that the enzyme cystathionine ~- normal levels
was associated with a 3. 4-fold increase had reported synthase was
lacking in a liver biopsy specimen from in risk of acute myocardial
infarction. Studies from another patient with homocystinuria. This
was the original Framingham Heart Study cohort (USA) the first
indication of a vitamin relationship to have shown strong, positive
correlation between homocystinuria, because that enzyme has as its
co- plasma homocysteine concentration and degree of factor vitamin
B6 (pyridoxal phosphate). Thereafter, carotid stenosis.
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