The basic mechanism underlying directional differences in
excitability, con- duction velocity, andsafetyfactor
thatleadtocircusmovementreentry incardiac muscle is generally
attributed to a spatial difference in the refractory period as
originally described by Mines [1] or to a depressed segment as
described by Schmitt and Erlanger [2]. A departure from this
depolarization in cardiac muscle involve quantities, such as Vmax'
that are not directly descriptive of the underlying mechanisms of
propagation.
General
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