The function and life span of endothelial cells have a large impact
upon the quality and expectancy of an individual's life. During low
perfusion, the adaptation of different cells to hypoxia precipitate
the aggressive progression of diseases. Although the clinical
studies have convincingly shown that endothelial dysfunction occurs
whenever the biological functions or bioavailability of nitric
oxide are impaired, in all these scenarios, the role of endothelial
cell-destructive process cross-talk is yet poorly understood. This
book focuses on the contribution of molecular mechanisms to
endothelial dysfunction in related metabolic disorders.
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