"Clostridium difficile" has been recognized as the cause of a broad
spectrum of enteric disease ranging from mild antibiotic-associated
diarrhea to pseudomembranous colitis. This volume gives new
insights into the microbiology, diagnostics and epidemiology of
"Clostridium difficile" and describes recent strategies in
treatment of diseases caused by this agent. Main parts of the
volume are devoted to "Clostridium difficile" toxins A and B which
are the major virulence factors. The molecular biology,
biochemistry, pharmacology and cell biology of these toxins which
are the prototypes of a new family of large clostridial cytotoxins
is described in great detail. "Clostridium difficile" toxins act as
glucosyltransferases to inactivate small GTP-binding proteins of
the Rho family which are involved in regulation of the actin
cytoskeleton, cell adhesion and various signaling processes.
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