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Books > Medicine > Clinical & internal medicine > Diseases & disorders > Oncology
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Mechanisms in B-Cell Neoplasia 1988 - Workshop at the National Cancer Institute, National Institutes of Health, Bethesda, MD, USA, March 23-25, 1988 (Paperback, Softcover reprint of the original 1st ed. 1988)
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Mechanisms in B-Cell Neoplasia 1988 - Workshop at the National Cancer Institute, National Institutes of Health, Bethesda, MD, USA, March 23-25, 1988 (Paperback, Softcover reprint of the original 1st ed. 1988)
Series: Current Topics in Microbiology and Immunology, 141
Expected to ship within 10 - 15 working days
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The papers in this book were presented at the 6th Workshop on
Mechanisms in B-Cell Neoplasia, held in Bethesda, March 23-25,
1988. On alternate years this meeting is sponsored by the . ;.
Basel Institute of Immunology in Basel, Switzerland and by the
National Cancer Institute in Bethesda, and is attended by 100 to
150 parti cipants. This 6th workshop, like the preceding five, was
characterized by intense and enthusiastic discussion which
reflects, we think, the exciting growth and development of this
field. It is quite clear, however, that despite many general
advances an understanding of the precise underlying mechanisms in
B-cell tumor development is not yet defined. Probably, there is no
single mechanism for all the various forms of B-cell neo plastic
development. Many different forms of B-cell neoplasms are known,
and these are distinguished by several characteristics: 1) the
stage of development attained by the tumor stem cells; 2) mode of
growth (slow or fast); 3) association with natural or inductive
etiologic agents and 4) specific and consistent mutational
mechanisms such as retroviral insertion, chromosomal rearrangement.
Those charac teristic forms which arise naturally in relatively
high frequency or those tumors with hallmark properties which can
be induced consistently are the models most frequently studied, e.
g. , endemic Burkitt's lymphoma, follicular lymphoma, acute and
chronic lymphocytic leukemia and mUltiple myeloma in man; bursal
lymphoma in chickens; Abelson virus induced pre B cell lymphomas
and plasmacytomas in mice and immunocytomas in rats. Each model
system, has special problems and advantages.
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