Numerous improvements in our understanding of the mechanisms
that underlie neuropathic pain states have come from the
development of animal models, most of which involve partial
peripheral nerve injury. The animal models have shown that nerve
injury initiates a cascade of events resulting in altered
neurochemistry and molecular biology of the peripheral neurons, the
dorsal root ganglion cell, and changes in neurotransmitter and
receptor expression in the dorsal horn of the spinal cord.
Moreover, nerve injury produces anatomical changes with functional
consequences.
This volume summarises the current understanding of the
pathophysiological processes in the peripheral and central nervous
system that contribute to the neuropathic pain. It provides a
timely review of neuropathic pain mechanisms, written by experts in
the field.
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