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Books > Science & Mathematics > Biology, life sciences > Life sciences: general issues > Neurosciences
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Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy (Paperback, Softcover reprint of the original 1st ed. 1993)
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Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy (Paperback, Softcover reprint of the original 1st ed. 1993)
Series: Advances in Experimental Medicine and Biology, 341
Expected to ship within 10 - 15 working days
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This volume contains the papers presented in the International
Summer Course on "Cirrhosis, Hyperammonemia and Hepatic
Encephalopathy," which was one of the prestigious Summer Course, of
the Complutense University of Madrid held in EI Escorial, Spain,
during August 10-14, 1992. Liver cirrhosis is one of the main
causes of death in western countries. In addition there is a series
of liver dysfunctions including fulminant hepatic failure, Reye's
syndrome and congenital defects of urea cycle enzymes that could
lead to hepatic encephalopathy, coma and death. As a consequence of
impaired liver function, the ability to detoxify ammonia by its
incorporation into urea is diminshed, resulting in increased
ammonia levels in blood and brain. Hyperammonemia is considered one
of the main factors in the mediation of hepatic encephalopathy and
the classical clinical treatments are directed towards reducing
blood ammonia levels. A part of the book is therefore devoted to
the study of certain aspects of ammonia metabolism such as the
regulation of the urea cycle, the main mechanism of ammonia
detoxification in mammals, which is located mainly in the liver.
The metabolism of ammonia in other tissues, including brain, is
also presented, as well as the effects of hyperammonemia on brain
metabolism and function and on brain microtubules. The control of
cerebral protein breakdown is reviewed. The classical and some
recently proposed clinical treatments as well as nutritional
considerations in the management of patients with liver failure are
also discussed.
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